Animal studies confirm that the relief some of us get from eating sugar is not just psychological--- it is an actual brain-chemistry reaction. In one experiment, Blass and colleagues studied two groups of baby mice who were separated from their mothers and left alone for six minutes. Their resulting “isolation distress” was considered to be a kind of animal equivalent to our human version of depression. The depressed mice who were given sugar water cried only seventy-five times during their isolation--- as compared to the more than three hundred cries that came from the mice left alone with no sweet treat to alleviate their emotional pain. Apparently, the young mice were literally “medicating” their depression with sugar.
Why did sugar have this remarkable effect? Researchers thought that perhaps the sweet food stimulated the release of extra beta-endorphin molecules. Since these molecules help us cope with physical and emotional pain, the sugar had a literally soothing effect. Researchers confirmed their theory by giving both groups of mice Naltrexone, a drug that blocks beta-endorphin receptors. If you take Naltrexone, it does not matter how many beta-endorphins you release--- you will not get any relief from pain. Sure enough, when the sugar-fed mice were given Naltrexone, they lost all interest in the sweet substance, suggesting that their only reason for their sweet tooth had been to stimulate the release of beta-endorphins.
`Numbed by Naltrexone, both groups of mice cried equally often. The poor baby mice were still depressed--- but now even sugar could not make them feel better.
翻譯:
動物實驗證實,某些人吃甜食會感到心情放鬆不只是心理因素─這的確是一種腦部化學反應。在一次實驗中,巴斯和他的同事研究兩組幼鼠。這兩組幼鼠與母親分開並獨處六分鐘。結果這些幼鼠會產生“隔離憂鬱”,也就是被視為相當於人類憂鬱症的一種動物憂鬱症。有餵食糖水的憂鬱幼鼠在隔離期間只哭了75次。作為對照組的幼鼠並未餵食糖水來緩和情緒上的傷痛,在隔離期間哭了超過三百次。顯然這些幼鼠確實是以糖當作為藥物,治療牠們的憂鬱症。